High Blood Pressure and Kidney Disease

Blood pressure measures the force of blood against the walls of your blood vessels. Blood pressure that remains high over time is called hypertension. Extra fluid in your body increases the amount of fluid in your blood vessels and makes your blood pressure higher. Narrow or clogged blood vessels also raise your blood pressure.
High blood pressure makes your heart work harder and, over time, can damage blood vessels throughout your body. If the blood vessels in your kidneys are damaged, they may stop removing wastes and extra fluid from your body. The extra fluid in your blood vessels may then raise blood pressure even more. It's a dangerous cycle.
High blood pressure is one of the leading causes of kidney failure, also commonly called end-stage renal disease (ESRD). People with kidney failure must either receive a kidney transplant or go on dialysis. Every year, high blood pressure causes more than 25,000 new cases of kidney failure in the United States.
Every year, high blood pressure causes more than 25,000 new cases of kidney failure in the United States.
Chronic kidney disease (CKD) is present when the estimated glomerular filtration rate (eGFR) is below 60 milliliters per minute (mL/min).
More than 30 milligrams of albumin per gram of creatinine in a urine sample is another sign of CKD.
People with CKD should try to keep their blood pressure below 130/80.
Two groups of medicines called angiotensin-converting enzyme (ACE) inhibitors and angiotensin receptor blockers (ARBs) lower blood pressure and have an added protective effect on the kidneys.
African Americans are six times more likely than Caucasians to develop hypertension-related kidney failure.
Early management of high blood pressure is especially important for African Americans with diabetes.

A New Breakthrough of FSGS Treatment

FSGS is short for Focal Segmental Glomerulosclerosis, which reflects the damage in the renal glomerulus. Due to the immune reaction caused by immune complex deposition, the damage in the kidney glomerulus are really serious gradually. The sclerosis will take place partially in the renal glomerulus. In this case, the blood and oxygen in the kidney will be insufficient, which in turn worsen damage in the kidney. In FSGS stage, quiet a few patients are under dialysis, which many people have realize that can not treat from root and with time passing by, patients may can not get rid of dialysis. Is there a new breakthrough of FSGS treatment?
The combination therapy of both western medicine and Chinese herbs will give a turn in the progressing of FSGS and make a breakthrough.
As we know, in many countries, kinds of hormones and immunosuppressive agents are applied, which can control the immune reaction in the kidney and diminish the symptoms. However, immune complex still exist in the kidney, which will lead to continuous reaction. Especially after infection, a lot of immune complex will appear, which causes symptom to appear once more.
So if we want to make a new breakthrough, we should enhance the immune ability as well as clear away the immune complex. To achieve this, Micro-Chinese Medicine Osmotherapy can take effects, which is partial immunotherapy in Kidney Disease Hospital.
Firstly, it can help extend the blood vessels especially the micro-blood vessels, in which case, the Focal Segmental Glomerulosclerosis can be relieved for the nutritious herbs can supply more oxygen and nutrition to the renal glomerulus. At this moment, the damaged cells can be repaired to some extent.
On the other hand, in Micro-Chinese Medicine, there are herbs which can enhance the immunity of patients with FSGS. In this case, our body gains ability to clear away the immune complex in the kidney. Even in the later time, infection invades, the immune system cells can defeat the bacteria and virus, etc, which can prevent the relapse of symptoms such as hematuria and proteinuria, etc. For more information about the new breakthrough of FSGS, welcome to communicate with us online or email us.

Polycystic kidney disease complicated by urinary tract infection, hematuria how to do?

What are the complications of polycystic kidney disease, polycystic kidney disease incidence is rising, sometimes bleeding suddenly occurs, can cause pain, bleeding from the cyst walls of cancer associated with cyst. In the positive treatment of polycystic kidney disease, while very effective to prevent, inhibit the occurrence of complications is very important. Polycystic kidney complications which it:
1, urinary tract infection: polycystic kidney complications including cystitis, pyelonephritis, cysts, infections and renal abscess. Women, urinary tract devices check their risk factors. Polycystic kidney complications appear leukocyte urinary tube and conventional anti-infection treatment response quickly favor the diagnosis of pyelonephritis positive blood culture and local tenderness tend cyst infection, CT can be found in infected cyst.
2, kidney stones and kidney calcification: polycystic kidney complications when the pain intensified, combined stones may twist infertility or intraocular hematuria should think.
3 cancerous cyst: cyst whether cancerous so far the issue remains controversial. When the pain the polycystic kidney complications in patients with kidney area or (and) hematuria law changes, weight loss, ESR increase, there does not comply with renal anemia, or polycythemia, polycystic kidney complications should be alert combined kidney cancer, for CT examination in a timely manner, if necessary, need for renal arteriography.
And close to the lower pole ureter cyst in the kidney, polycystic kidney complications aggravated hydronephrosis, and the oppression of the urine of the renal pelvis can cause back pain. Polycystic kidney complications, such obstruction can also kidney infection.

Female patients with polycystic kidney pregnant, will be passed on to children?

Polycystic kidney disease is a genetic kidney disease, polycystic kidney disease increase with age, the cysts gradually increased, the first performance: lower back pain, high blood pressure, abnormal renal B results, if not timely control kidney cysts continue to increase, and oppression around the renal unit will cause injury to start the process of renal fibrosis, will show as urine tests appear: leakage of protein and blood cells; renal fibrosis process has not been effective in deterring appears elevated serum creatinine or glomerular filtration rate, and ultimately develop into end-stage renal failure.
The nephropathy experts, polycystic kidney disease is not only influenced by genetic factors, and the age of the patient at the same time, the geographical location is also certain. Hereditary polycystic kidney disease can be divided into two types: adult polycystic kidney disease is autosomal dominant infantile polycystic kidney disease is autosomal recessive.
Numerous cysts polycystic kidney Department of the renal cortex and medulla of a hereditary disease. The kidney specialists pointed out that the mode of inheritance can be divided into autosomal dominant polycystic kidney and autosomal recessive polycystic kidney disease.
Adult polycystic kidney disease Genetic incidence of approximately 0.2%, mainly in the kidney cysts, increased development and the number of characterized, often accompanied by other organs cyst formation, most common liver, pancreatic, ovarian, and gastrointestinal tract can also be seen.
Autosomal recessive polycystic kidney genetic rule is: both parents heterozygous and homozygous offspring incidence, the incidence probability of 25%. Gender and racial differences.
The etiology of polycystic kidney disease autosomal dominant polycystic kidney genetic laws: female incidence probability thought; either parent for sick children 50% of cysts gene and the disease, if the parents are sick, the increase in the incidence of children to 75%; sick children do not carry the cysts genes.
So, polycystic kidney disease genetic risk is still high, there are the polycystic kidney women for our children's health, it is best not pregnant.

Can kidney disease patients eat fish

Abalone is a relatively expensive food possible life we do not often edible, the abalone indeed have a lot of nutritional value, and are very helpful for the treatment of many diseases. Nutrition experts pointed out: abalone is the best food for kidney. Conditional friends, especially those who suffer from kidney disease friends, regular consumption of abalone to the kidney.
Abalone is a relatively expensive food possible life we do not often edible, the abalone indeed have a lot of nutritional value, and are very helpful for the treatment of many diseases. Nutrition experts pointed out: abalone is the best food for kidney. Conditional friends, especially those who suffer from kidney disease friends, regular consumption of abalone to the kidney.
Material: skinless chicken subcutaneous fat, 80 grams, 25 grams of abalone.
Method: peeled and subcutaneous fat of chicken and abalone, plus adequate water into the cooker, first boil over high heat, then simmered in boiling down. Add a little salt to taste (be careful not to salt too much), soup with meat taking food.
Effect: Chicken natured, sweet and salty, rich in high quality protein, functions tonic temperature benefit internal organs, can cure a weak stomach, liver and kidney tonic sinister. Medicine to non-spawning female chicken is better, the best choice of Silky, most nourishing skill. Abalone seafood molluscs, flat, sweet and salty, rich in protein function Yin and kidney, smart head. Therefore, we look at the role of the therapeutic side, main role of Bushentianjing.
Indications: The side taste delicious, easy to cause the patient appetite, rich in high quality protein, of hypoproteinemia nephrotic syndrome, benefit the therapeutic side applies to patients with membranous nephropathy with nephrotic syndrome, edema after treatment has is not serious, but still mild to moderate proteinuria, plasma protein, renal function is still good, those without elevated serum creatinine or blood urea nitrogen (azotemia).
Contraindications: nephrotic syndrome associated with severe azotemia, impaired renal function, unfit for human consumption.

What is the reason for anemia with ESRD patinets

The main component of RBC hemoglobin. Hemoglobin is composed of protein and iron in the synthesis process also requires maintenance bovine hormone c and Vitamin B: participation. Bone alkaline place of the red blood cell synthesis. Entire generation process of the red blood cells, including the synthesis of hemoglobin, the red blood cells of mature kidneys secrete erythropoietin factor and red blood cell known as a very close ... to generate ESRD anemia following detailed the introduction:
The main component of RBC hemoglobin. Hemoglobin is composed of protein and iron in the synthesis process also requires maintenance bovine hormone c and Vitamin B: participation. Bone alkaline place of the red blood cell synthesis. Entire generation of red blood cells, including the synthesis of hemoglobin, red blood cells mature, with the kidneys secrete erythropoietin factor and the red blood cells to generate known as a very close relationship. Specifically, the ESRD anemia mainly due to the following aspects.
(1) lack of nutrition: reduce chronic renal failure patients with long-term low-protein diet, protein synthesis in the body, but there are large numbers of protein loss in the urine, together with patients with symptoms of anorexia. Intestinal absorption function is not a good result, blood raw ( such as iron, folic acid, protein) intake. These factors will inevitably patients with malnutrition leading to anemia.
(2) erythropoietin decreased: in ESRD, causing serious damage to the renal parenchyma, the kidney erythropoietin factor and erythropoietin reduce erythropoietin stimulation of bone marrow was weakened red blood cell production and mature obstacles therefore appear progressive anemia.
Accelerated destruction of red blood cells (3): chronic renal failure, the body of metabolic wastes (such as methyl clothing) from the urine decreased. Concentration in the blood, such substances can accelerate the destruction of red blood cells, the red blood cells shortened life expectancy . caused hemolysis.
(4) chronic blood loss: melons substances and phenols in the blood of patients with renal failure can not be from the urine, one hand, these substances can cause coagulation abnormalities; They also enable the wool sewing vascular fragility. Because of the role of these toxic substances, many patients with end stage renal disease often epistaxis, teeth bowel bleeding, subcutaneous bleeding and gastrointestinal bleeding. These also cause anemia reasons. Anemia caused by renal parenchymal disease called renal anemia. The nature of iron deficiency anemia or small cell hypochromic anemia. More severe renal failure, anemia is more powerful.

Hypertension will cause kidney failure

15% of the high blood pressure will develop renal failure, and this is because the body of the urinary system is like one; wastewater excretory system, plays an important role in maintaining the machine physiological equilibrium process. The kidney is an important organ of the urinary system, it is like the central part of the water treatment system, the kidney is good or bad on the body is very important. Harm to the kidneys addition to allergic inflammation caused by bacterial infection, the more common clinical hypertension.
Under normal circumstances, hypertension involving the kidney is a relatively lengthy process. Pathological studies have shown that hypertensive kidney damage, mainly from the arterioles, no obvious early on renal morphology and function of the change. First, the renal artery hardening, stenosis, renal ischemic, some nephron fibrosis hyalinization, the normal nephron compensatory hypertrophy, with the continuous development of the disease, the surface of the kidney was granular, cortical thinning and shrinking due to the repeated destruction of the nephron, the kidney, and subsequent renal dysfunction and the development of uremia. However, due to the strong compensatory ability of the kidney to begin the only symptoms of renal dysfunction is nocturia increased. Urine routine examination, it may be seen under a microscope the red blood cells, of proteinuria and tube type.
Development decompensated renal insufficiency, concentrating ability of the kidneys to reduce the symptoms of polyuria, thirst, polydipsia, urine specific gravity lower and fixed around 1.010. When the further development of renal insufficiency, urine output decreased significantly, the blood of non-protein nitrogen, creatinine, urea nitrogen levels, edema, electrolyte imbalance and acid-base balance, X-ray or B ultrasound examination showed bilateral renal symmetrical mild narrowing. Selective renal arteriography may have varying degrees of renal artery stenosis.